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Wherein, the phosphorylation of the C-terminus by ERK1/2 recruits β -arrestins to sterically hinder interactions with GPCR kinases, initiating endocytosis likely via the assembly of clathrin-coated pits [24, 33]
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Indeed, hepatic GCGR signaling is essential for fatty acid oxidation as well as hepatic triglyceride synthesis and secretion in mice [51]
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