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However, while our results corroborate the existence of GLP-1R-associated endosomal signalling, they also suggest that sustained GLP-1R signalling, as indicated by cumulative insulin secretion over pharmacologically relevant timescales, is actually greater with the ligand (exendin-4-C16) with a reduced tendency to recruit mini-G s to Rab5-positive endosomes
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Thus, are changes in the gut bacterium itself beneficial to the host
