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Interestingly, administration of liraglutide in mice with selective disruption of the cardiomyocyte GLP-1R ( Glp1r CM−/ − ) generated from MerCreMer transgenic mice expressing tamoxifen-inducible Cre driven by the α-myosin heavy chain promoter, still produced robust cardioprotection and increased survival, suggesting that cardiomyocyte GLP-1R activation is not required for adaptive responses to ischemic and cardiomyopathic injury mediated by liraglutide (Ussher et al
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doi: 10.1080/01621459.1989.10478874 47 AustinPC
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