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DM has been shown to significantly disrupt the expression of key components in this metabolism pathway, leading to decreased lactate availability and energy supply to germ cells (19–22)
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PHARMACOLOGY OF GLP-1 RAs Members of the GLP-1 RA class mimic the action of endogenous GLP-1: they stimulate insulin secretion from pancreatic beta-cells in a glucose-dependent manner, and, during hyperglycemia, GLP-1 RAs reduce inappropriately elevated levels of glucagon, which results in decreased hepatic glucose output
